What is the purpose of using anticoagulants in stroke patients?

Using anticoagulants in stroke patients primarily serves to prevent clot formation in those who have specific risk factors. Conditions such as atrial fibrillation, deep vein thrombosis, or a history of prior strokes increase the likelihood of thrombus formation, which can lead to ischemic strokes. Anticoagulants work by interrupting various steps in the clotting process, thereby reducing the likelihood of new clots developing.

Preventing clot formation is crucial in managing stroke risk because it helps maintain blood flow to the brain and reduces the incidence of recurrent strokes. In particular, patients with a high risk of blood clots are placed on anticoagulants as a proactive measure to safeguard brain health and overall well-being.

While other treatments such as thrombolytics exist to dissolve existing clots, these are not the same as anticoagulants, which focus on prevention rather than dissolution. Reducing blood pressure and enhancing blood flow through vessel dilation are also important in overall stroke management but do not specifically relate to the direct action of anticoagulants.